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As the primary active component in ginseng, Ginsenoside Rg3 (GRg3) has been shown to inhibit the excessive release of pro-inflammatory factors by modulating the TLR4/Myeloid Differentiation Primary Response 88 (MyD88)/NF-B pathway

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Without proper medical guidance, this increases the risk of both ineffectiveness and unintended health consequences [47]

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