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Research has demonstrated that UA intervention decreases ROS production by inhibiting the activity of NADPH oxidase, a key source of cellular ROS, and subsequently reduces the production of proinflammatory mediators through the regulation of the phosphatidylinositol 3-kinase / protein kinase B / nuclear factor kappa B and c-Jun N-terminal kinase / activator protein 1 signaling pathways (59)

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It has shown promising results in suppressing the growth and proliferation of malignant blood cells

Skin renewal activity of non-thermal plasma through the activation of beta-catenin in keratinocytes