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Peptide therapy for inflammation represents a paradigm shift in how we approach chronic pain and tissue damage

This process involves the disruption of cadherin-based cellcell contacts and subsequent cell motility, and is a key epithelial function in embryogenesis and wound repair [Corso et al

As a person ages, the level of homocysteine in the blood increases, leading to the development of dementia

These platforms must be adaptable not only to the biochemical signals of infection and inflammation but also to mechanical, enzymatic, and microbial stressors, particularly in complex wounds, diabetic ulcers, and infection-related cancers