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2022;23(8):e52280 91

Robust patients (non-frail and non-sarcopenic) had the highest survival rates, followed by non-frail sarcopenic patients

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Using SLE as an example below, we discuss several potential biologic pathways involving epigenomics, the microbiome, and immune dysregulation that lead to inflammation and organ damage, mechanisms that may also apply to the development of other SARD ( Figure 1 )