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When the body is under acute or chronic stress, it often reduces the action of the thyroid hormone as a defensive strategy to preserve electricity [26]

In prenatally hypoxic rats, being treated with nMitoQ could suppress the rise in ROS and attenuate the heart diastolic dysfunction, especially in female progeny 514 Furthermore, improved cardiac tolerance after adulthood could be seen in the offspring of these prenatal hypoxic pregnant rats administrated with nMitoQ

FTH1, ferritin heavy chain 1

77,83,84 In addition, the expression of ALDH1L2 is upregulated by some certain drugs, such as thapsigargin and tunicamycin, endoplasmic reticulum stress inducers in immortalized human B cells, 85 mitotane, an adjuvant monotherapy used for treating adrenocortical carcinoma, 86 and the indomethacin, an anti-inflammatory agent in breast cancer cells