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In general, the term can also be used in a broad sense, meaning any such chromatic or morphological alteration, localized and persistent, present on the skin surface

These include use of immunomodulatory agents as well as inhibitors of B-cell receptor signaling that target either Bruton tyrosine kinase (ibrutininb) or PI3 kinase delta (idelalisib).50 Pro-oxidant based therapies utilize oxidative stress as a cellular weakness of leukemic cells and represent an example of a synthetic lethality approach

With the help of cell cycle arrest, DNA repair pathways are subsequently activated to inhibit cell cycle progression, providing a window of repair for the cell

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