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Abstract Nuclear factor erythroid 2related factor 2 (Nrf2) is a key transcription factor that regulates the expression of genes involved in cellular protection against oxidative stress and the maintenance of redox homeostasis

The present report indicates that the earliest damage found in the liver after diquat administration is endothelial cell injury

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L-Glutathione (GSH), especially in nanoparticle slow-release form, maintains redox tone but excessive co-delivery creates a reductive stress, paradoxically inactivating Nrf2, SIRT3, and AMPK, the very genes responsible for antioxidant renewal