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360 At the cellular level, excess of ammonium mainly affects astrocytes with neuronal injury being secondary, resulting mainly from failure of astrocytic homoeostatic cascades and oedema

Boredom-Based e-Learning System using EEG, Isha Ghodgaonkar, Nathalia Peixoto 45

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However, if STAT5b mediates both metabolic signaling and IGF-I production it still needs to be understood where the two pathways diverge, and why GH metabolic signaling is not blocked in the absence of insulin crosstalk