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reported an increase in the levels of dysfunctional mitochondria and mitochondrial membrane proteins, translocase of the outer mitochondrial membrane 20 (Tom20), translocase of the inner mitochondrial membrane 23 (Tim23), and porin in the ASD temporal cortex without a change in the transcription of genes, such as PARK2 , responsible for the activation of mitophagy

Facing the same problems over the last 8 years we have developed a different approach (4.6): (1) delivering drugs to cerebral cortical target sites via a "transmeningeal" vs