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SELENOS has a role in the removal of stressor-induced misfolded proteins from the ER, preventing the accumulation of these proteins and the subsequent stress response that leads to activation of NF-B, pro-inflammatory cytokine gene transcription and the inflammation cascade (Fig

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Enzyme-responsive biomimetic ferritin nanoparticles (MMFn) engineered with MMP-2/9-responsive peptides demonstrated robust antitumor activity in Panc02 pancreatic cancer xenograft models without systemic toxicity (122)

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