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Bhattacharjee and Borah found that mitochondrial dysfunction and the development of oxidative stress could be the potential mechanisms for homocysteine-induced degeneration of dopaminergic neurons in the SN in the rat model of PD (Bhattacharjee and Borah 2016)

Emerging studies on post-viral syndromes highlight the role of neuroinflammation, where the brain's resident immune cells (microglia) become chronically activated

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