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doi: 10.7759/cureus.78361 Arif Sehrish

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Point mutations of both Cqd1 and Cqd2 at conserved protein-kinase-like (PKL) or UbiB motif residues disrupted their individual impact on CoQ 6 distribution, indicating that Cqd1 and Cqd2 activities rely on atypical kinase/ATPase activity, though further biochemical work is needed to prove enzymatic activity