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5-Azacitidine is modified by nucleotide reductase and then phosphorylated by uridine cytidine kinase to form a triphosphate, 5-azacitidine triphosphate, which is integrated into the DNA sequence as a cytosine analog during DNA replication, thereby inhibiting DNA cytosine methylation (Rius et al., 2009)

Lee S, Kim SM, Lee RT (2013) Thioredoxin and thioredoxin target proteins: from molecular mechanisms to functional significance

It was identified that as the degree of ferroptosis increased, the proportion of TUNEL-positive cells significantly increased, suggesting that ferroptosis promoted HK-2 cell death under a high concentration of CaOx (Fig

doi: 10.1016/j.mehy.2004.03.021