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In addition, Irg1-deficient phagocytes, which cannot produce itaconate, have shown increased susceptibility to M.tb infection, with infected mice exhibiting higher bacterial loads, reduced survival, exacerbated inflammation, extensive necrotizing granuloma formation and elevated neutrophil infiltration in the lungs [104, 105]

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Establishing appropriate antibiotic dosages facilitates a clearer understanding of the dynamics between bacterial pathogens and antimicrobial agents, paving the way for future studies aimed at elucidating the intricate interactions that govern antibiotic efficacy and resistance