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Zhu G, Guan F,Li S, Zhang Q, Li Y

Neurons are particularly vulnerable due to high PUFA content (DHA, arachidonic acid) and mitochondrial dysfunction that disrupts coenzyme Q10 synthesis (100102)

Accelerated senescence in animal models has been decelerated by carnosine administration accompanied by decreased activities of Monoamine oxidase (MAO, a flavin-containing mitochondrial enzyme) and Na + /K + -ATP-ase in synaptosome (Yuneva et al