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Their FAO suppression leads to reduced fatty acid breakdown and lipid accumulation, forming lipid-laden macrophages. These macrophages have severely impaired phagocytic function and cannot effectively clear apoptotic cells, foreign bodies, and cigarette smoke particles, further exacerbating the stability of the inflammatory microenvironment

For example, in studies of men with high burden relapsed metastatic prostate cancer, CTC count included in a composite panel with serum lactate dehydrogenase is a surrogate of survival and captures the effect of ARSI treatment [118]

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Who may not be a good candidate Peptide therapy may not be appropriate for everyone