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Epigenetic modifications not only underlie the molecular mechanisms driving disease development but also serve as promising biomarkers for clinical diagnosis and enable precision-targeted therapeutic interventions (31,32)

Li A, Gao M, Liu B, Qin Y, Chen L, Liu H, et al

Nassireslami et al., 2016)

Comparative metabolomic profiling of cystine-repleted or -starved NSCLC cells revealed that cystine-starved cells accumulate -glutamyl-di- or tripeptides, which were produced by non-canonical activity of GCLC