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Human data outside the trial setting remain limited, and standalone long-term safety has not been established since the compound is still investigational

These findings suggest that elevated p53 levels cause ferroptosis, whereas p21 can act as an inhibitor of ferroptosis

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Recent evidence suggests that, even among hyperthyroid patients, maintaining an adequate dietary iodine intake improves therapeutic outcomes, enhancing response to treatment, reducing recurrence, increasing remission rates, and providing better control of thyrotoxicosis compared with inadequate or excessive iodine intake [40,41,42]