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Given the hypothesis proposed regarding reduced glutathione biosynthesis around this timeframe within our study, conversion of excess GLY to other amino acids offers a plausible explanation

Luo and Zhang, 2011)

As TNF-ais a proinammatory molecule known to be induced by stimuli common in the lungs of patients with CF, such as bacterial LPS, the possibility of blocking these effects by a drug already in use for long-time treatment in CF is relevant, and indicates that other TNF-aregulated genes beside GSTT1 and GSTM1 might be affected by AZM

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